Abstract
Using the hairless mouse ear skin flap model we demonstrate that HSP-32 expression has to be considered as a significant mechanism of ischemic preconditioning-induced protection of critically perfused tissue.
MeSH terms
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Animals
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Blood Flow Velocity / physiology
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Graft Survival / physiology*
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Heme Oxygenase (Decyclizing) / physiology*
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Heme Oxygenase-1
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Ischemic Preconditioning / methods*
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Laser-Doppler Flowmetry
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Membrane Proteins
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Mice
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Mice, Hairless
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Microcirculation / physiopathology
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Microsurgery
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Surgical Flaps / blood supply*
Substances
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Membrane Proteins
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Heme Oxygenase (Decyclizing)
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Heme Oxygenase-1
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Hmox1 protein, mouse