Phorbol ester-induced secretion of human hepatocyte growth factor by human skin fibroblasts and its inhibition by dexamethasone

FEBS Lett. 1992 Apr 13;301(1):107-10. doi: 10.1016/0014-5793(92)80220-b.

Abstract

Human skin fibroblasts secreted a certain amount of human hepatocyte growth factor (hHGF), as determined by an enzyme-linked immunosorbent assay for hHGF. This hHGF secretion was remarkably stimulated by protein kinase C (PKC)-activating phorbol esters, which was inhibited by the simultaneous addition of dexamethasone. Pretreatment with phorbol 12-myristate 13-acetate (PMA) caused a down-regulation in hHGF secretion. hHGF secreted by the PMA-treated cells showed a potent hepatocyte growth-promoting activity which was neutralized by an anti-hHGF antiserum. These results indicate both that PMA-treated human skin fibroblasts produce biologically active hHGF and the possible involvement of PKC activation in this process.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Dexamethasone / pharmacology*
  • Dose-Response Relationship, Drug
  • Down-Regulation
  • Enzyme Activation
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Fibroblasts / drug effects
  • Fibroblasts / metabolism*
  • Hepatocyte Growth Factor / metabolism*
  • Humans
  • Infant, Newborn
  • Protein Kinase C / metabolism
  • Skin / cytology
  • Skin / drug effects
  • Skin / metabolism
  • Tetradecanoylphorbol Acetate / pharmacology*

Substances

  • Hepatocyte Growth Factor
  • Dexamethasone
  • Protein Kinase C
  • Tetradecanoylphorbol Acetate