The role of lipoproteins in the inactivation of endotoxin by serum

Infusionsther Transfusionsmed. 1992 Aug;19(4):202-3. doi: 10.1159/000222624.

Abstract

The lipopolysaccharide (LPS)-induced release of cytokines from human monocytes can be prevented by previous interaction of serum with LPS. This inactivation is a function of lipoproteins. Here we show that LPS can be inactivated by low-density lipoproteins (LDL) as well as by high-density lipoproteins (HDL). The effects of heparin and EDTA on LPS inactivation by serum are also described.

MeSH terms

  • Bacterial Toxins / immunology*
  • Dose-Response Relationship, Drug
  • Edetic Acid / pharmacology*
  • Endotoxins*
  • Enterotoxins / immunology*
  • Heparin / pharmacology*
  • Humans
  • Lipopolysaccharides / immunology*
  • Lipoproteins / pharmacology*
  • Lipoproteins, HDL / pharmacology
  • Lipoproteins, LDL / pharmacology
  • Lipoproteins, VLDL / pharmacology
  • Monocytes / drug effects*
  • Monocytes / immunology
  • Monokines / physiology*

Substances

  • Bacterial Toxins
  • Endotoxins
  • Enterotoxins
  • Lipopolysaccharides
  • Lipoproteins
  • Lipoproteins, HDL
  • Lipoproteins, LDL
  • Lipoproteins, VLDL
  • Monokines
  • salmonella toxin
  • Heparin
  • Edetic Acid