Deletion mutagenesis of stem cell factor defines the C-terminal sequences essential for its biological activity

Biochem Biophys Res Commun. 1992 Oct 15;188(1):292-7. doi: 10.1016/0006-291x(92)92383-9.

Abstract

We constructed a series of murine stem cell factor (mSCF) cDNAs which were sequentially truncated at the 3' termini. The resultant six mutant cDNA encode N-terminal 183, 179, 162, 149, 142 and 133 amino acid residues of the mature mSCF protein fused to the heterogeneous C-terminal peptides derived from the linker sequences. Each mutant cDNA was transiently expressed in COS cells, and the cultured supernatant was assayed for its ability to support the growth of a human factor-dependent cell line, TF-1 and to enhance colony formation by murine hematopoietic progenitor cells. The results showed that as few as N-terminal 142 but not 133 amino acid residues of mSCF remained biologically active in vitro, suggesting that the region of 9 amino acids from Asp134 to Ser142 containing a Cys138-mediated disulfide bond may contribute to the C-terminal end of the active subdomain of mSCF.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Base Sequence
  • Cell Division / drug effects
  • Cell Line
  • Hematopoietic Cell Growth Factors / genetics*
  • Hematopoietic Cell Growth Factors / pharmacology*
  • Hematopoietic Stem Cells / cytology*
  • Hematopoietic Stem Cells / drug effects
  • Humans
  • Kinetics
  • Mice
  • Molecular Sequence Data
  • Mutagenesis
  • Oligodeoxyribonucleotides
  • Polymerase Chain Reaction
  • Recombinant Proteins / pharmacology
  • Sequence Deletion*
  • Stem Cell Factor
  • Transfection

Substances

  • Hematopoietic Cell Growth Factors
  • Oligodeoxyribonucleotides
  • Recombinant Proteins
  • Stem Cell Factor