Abstract
A Ca(2+)-sensitive electrode was used to measure the Ca2+ concentration of the medium containing the heavy fraction of the fragmented sarcoplasmic reticulum (SR) prepared from guinea-pig psoas muscle. Among K(+)-channel blockers tested, 4-aminopyridine (4-AP), tetraethylammonium (TEA) and charybdotoxin elicited Ca2+ release from the SR, but apamin and glibenclamide did not. These results suggest that a reduction of SR K+ conductance leads to Ca2+ release from the SR.
MeSH terms
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4-Aminopyridine / pharmacology
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Animals
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Apamin / pharmacology
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Calcium / metabolism*
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Charybdotoxin
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Glyburide / pharmacology
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Guinea Pigs
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In Vitro Techniques
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Muscle Contraction / drug effects*
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Potassium Channels / drug effects*
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Psoas Muscles / drug effects
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Sarcoplasmic Reticulum / metabolism*
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Scorpion Venoms / pharmacology
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Tetraethylammonium
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Tetraethylammonium Compounds / pharmacology
Substances
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Potassium Channels
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Scorpion Venoms
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Tetraethylammonium Compounds
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Charybdotoxin
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Apamin
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Tetraethylammonium
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4-Aminopyridine
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Glyburide
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Calcium