The effect of nizatidine on neuromuscular transmission

In Vivo. 1992 Nov-Dec;6(6):597-600.

Abstract

The effect of the H2-receptor antagonist, nizatidine, on neuromuscular transmission was investigated using sciatic nerve-gastrocnemius muscle preparations of rat in vivo. Nizatidine, administered by i.v. injection, potentiates the neuromuscular blockade induced by d-tubocurarine, pancuronium and the aminoglycoside antibiotic, kanamycin. Moreover, the drug alone is capable of producing a blockade on preparations stimulated at high frequency. The neuromuscular blockade induced by nizatidine is reversed by 4-aminopyridine but not by dimaprit.

MeSH terms

  • 4-Aminopyridine / pharmacology
  • Animals
  • Dimaprit / pharmacology
  • Drug Synergism
  • Kanamycin / pharmacology
  • Male
  • Muscle Contraction / drug effects
  • Neuromuscular Junction / drug effects*
  • Neuromuscular Junction / physiology
  • Nizatidine / antagonists & inhibitors
  • Nizatidine / pharmacology*
  • Pancuronium / pharmacology
  • Rats
  • Sciatic Nerve / drug effects
  • Sciatic Nerve / physiology
  • Synaptic Transmission / drug effects
  • Tubocurarine / pharmacology

Substances

  • Kanamycin
  • 4-Aminopyridine
  • Pancuronium
  • Nizatidine
  • Tubocurarine
  • Dimaprit