Selective repression of c-fos gene transcription in rat embryo fibroblasts transformed by oncogenes E1A and cHa-ras

Biochem Biophys Res Commun. 2003 Jun 27;306(2):483-7. doi: 10.1016/s0006-291x(03)00996-3.

Abstract

Transformation of REF cells by oncogenes E1A and cHa-ras leads to activation of AP-1 factor concomitantly with down-regulation of c-fos gene transcription. Here we addressed two issues: (i) how does transcription of Fos/Jun-regulated genes change in the cells lacking Fos-Jun heterodimers; (ii) to which extent HDAC-mediated chromatin reorganization does affect, apart from c-fos, transcription of some other early and late-response genes. To this end, we studied the kinetics of serum-stimulated transcription of c-fos, c-jun, fra-1, egr-1, and cyclinD1 genes, as well as the effects of sodium butyrate, an inhibitor of histone deacetylase activity, on transcription of these genes in normal REF cells and transformants E1A+ras.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenovirus E1A Proteins / metabolism
  • Animals
  • Blotting, Western
  • Cell Line, Transformed
  • Cells, Cultured
  • Chromatin / metabolism
  • Dimerization
  • Fibroblasts / metabolism
  • Histone Deacetylases / metabolism
  • Kinetics
  • Proto-Oncogene Proteins c-fos / metabolism*
  • RNA, Messenger / metabolism
  • Rats
  • Reverse Transcriptase Polymerase Chain Reaction
  • Sodium Oxybate / pharmacology
  • Transcription, Genetic*
  • ras Proteins / metabolism

Substances

  • Adenovirus E1A Proteins
  • Chromatin
  • Proto-Oncogene Proteins c-fos
  • RNA, Messenger
  • Sodium Oxybate
  • Histone Deacetylases
  • ras Proteins