Characterization of angiotensin-(1-7) receptor subtype in mesenteric arteries

Peptides. 2003 Mar;24(3):455-62. doi: 10.1016/s0196-9781(03)00062-7.

Abstract

Mesenteric arteries from male Sprague-Dawley rats were mounted in a pressurized myograph system. Ang-(1-7) concentration-dependent responses were determined in arteries preconstricted with endothelin-1 (10(-7)M). The receptor(s) mediating the Ang-(1-7) evoked dilation were investigated by pretreating the mesenteric arteries with specific antagonists of Ang-(1-7), AT(1) or AT(2) receptors. The effects of Ang-(3-8) and Ang-(3-7) were also determined. Ang-(1-7) caused a concentration-dependent dilation (EC(50): 0.95 nM) that was blocked by the selective Ang-(1-7) receptor antagonist D-[Ala(7)]-Ang-(1-7). Administration of a specific antagonist to the AT(2) receptor (PD123319) had no effect. On the other hand, losartan and CV-11974 attenuated the Ang-(1-7) effect. These results demonstrate that Ang-(1-7) elicits potent dilation of mesenteric resistance vessels mediated by a D-[Ala(7)]-Ang-(1-7) sensitive site that is also sensitive to losartan and CV-11974.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Angiotensin I / pharmacology
  • Animals
  • Antihypertensive Agents / pharmacology
  • Endothelin-1 / pharmacology
  • Male
  • Mesenteric Arteries / metabolism*
  • Peptide Fragments / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Angiotensin / classification*
  • Receptors, Angiotensin / metabolism*
  • Vasodilation / drug effects

Substances

  • Antihypertensive Agents
  • Endothelin-1
  • Peptide Fragments
  • Receptors, Angiotensin
  • Angiotensin I
  • angiotensin I (1-7)