Induction of flexibility through protein-protein interactions

J Biol Chem. 2003 May 16;278(20):18581-7. doi: 10.1074/jbc.M300866200. Epub 2003 Feb 25.

Abstract

The dimerization/docking (D/D) domain of the cyclic AMP-dependent protein kinase (PKA) holoenzyme mediates important protein-protein interactions that direct the subcellular localization of the enzyme. A kinase anchoring proteins (AKAPs) provide the molecular scaffold for the localization of PKA. The recent solution structures of two D/D AKAP complexes revealed that the AKAP binds to a surface-exposed, hydrophobic groove on the D/D. In the present study, we present an analysis of the changes in hydrogen/deuterium exchange protection and internal motions of the backbone of the D/D when free and bound to the prototype anchoring protein, Ht31(pep). We observe that formation of the complex results in significant, but small, increases in H/D exchange protection factors as well as increases in backbone flexibility, throughout the D/D, and in particular, in the hydrophobic binding groove. This unusual observation of increased backbone flexibility and marginal H/D exchange protection, despite high affinity protein-ligand interactions, may be a general effect observed for the stabilization of hydrophobic ligand/hydrophobic pocket interactions.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptor Proteins, Signal Transducing*
  • Biochemical Phenomena
  • Biochemistry
  • Carrier Proteins / chemistry*
  • Carrier Proteins / metabolism
  • Humans
  • Hydrogen / chemistry
  • Kinetics
  • Magnetic Resonance Spectroscopy
  • Models, Molecular
  • Peptides / chemistry
  • Protein Binding*
  • Protein Structure, Secondary
  • Thermodynamics

Substances

  • Adaptor Proteins, Signal Transducing
  • Carrier Proteins
  • Peptides
  • ROPN1L protein, human
  • Hydrogen