Abstract
Neurologic complications from cerebral ischemia occur frequently following cardiac arrest, as well as in the perioperative period in cardiac surgery. The cellular and molecular mechanisms of cerebral ischemia are complex. This article discusses several important cell death and salvage pathways that are important in experimental cerebral ischemia that may be critical to outcome in clinical brain injury.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, P.H.S.
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Review
MeSH terms
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Animals
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Brain Ischemia / epidemiology
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Brain Ischemia / etiology*
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Brain Ischemia / physiopathology
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Cardiac Surgical Procedures
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Heart Arrest / epidemiology
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Heart Arrest / surgery
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Humans
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Incidence
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Postoperative Complications / epidemiology
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Postoperative Complications / etiology
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Postoperative Complications / physiopathology
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Stroke / epidemiology
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Stroke / etiology
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Stroke / physiopathology