Stroke damage in mice after knocking the neutrophin-4 gene into the brain-derived neurotrophic factor locus

J Cereb Blood Flow Metab. 2003 Feb;23(2):150-3. doi: 10.1097/01.WCB.0000043949.67811.C6.

Abstract

Neurotrophins play a protective role during cerebral ischemia, and mice lacking both alleles for neurotrophin 4 (Nt4-/- ) or deficient in a single allele for brain-derived neurotrophic factor (Bdnf+/-) have increased susceptibility to cerebral ischemia. This study directly compared the biologic activities of brain-derived neurotrophic factor (BDNF) and NT4 by replacing the coding sequence with the Nt4 sequence (Bdnf +/nt4-ki ). Mice expressing one allele in place of develop 61% bigger lesions after 1-hour middle cerebral artery occlusion compared with wild-type littermates. Physiologic parameters did not contribute to ischemia susceptibility. In conclusion, NT4 is less potent than BDNF in promoting brain survival after stroke.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Brain Ischemia / complications
  • Brain Ischemia / metabolism
  • Brain Ischemia / pathology
  • Brain-Derived Neurotrophic Factor / genetics
  • Brain-Derived Neurotrophic Factor / metabolism*
  • Cerebral Infarction / pathology
  • Mice
  • Mice, Transgenic / genetics
  • Nerve Growth Factors / genetics
  • Nerve Growth Factors / metabolism*
  • Nervous System Diseases / etiology
  • Stroke / complications
  • Stroke / metabolism*
  • Stroke / pathology*

Substances

  • Brain-Derived Neurotrophic Factor
  • Nerve Growth Factors
  • neurotrophin 4