Donor cardiac allografts from p53 knockout mice exhibit apoptosis-independent prolongation of survival

Transplant Proc. 2002 Dec;34(8):3274-6. doi: 10.1016/s0041-1345(02)03581-9.

Authors

M H Kown¹, S Murata, C L Jahncke, C Mari, G J Berry, M A Lijkwan, F G Blankenberg, H W Strauss, R C Robbins

Affiliation

¹ Department of Cardiothoracic Surgery, Stanford University, Stanford, California 94305, USA.

PMID: 12493444
DOI: 10.1016/s0041-1345(02)03581-9

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Animals
Apoptosis / immunology*
Caspase 3
Caspases / metabolism
Genes, p53*
Graft Rejection / pathology*
Graft Survival / immunology*
Heart Transplantation / immunology*
Heart Transplantation / pathology
Mice
Mice, Inbred BALB C
Mice, Inbred C57BL
Mice, Knockout
Tissue Donors
Transplantation, Heterotopic
Tumor Suppressor Protein p53 / deficiency
Tumor Suppressor Protein p53 / genetics*

Substances

Tumor Suppressor Protein p53
Casp3 protein, mouse
Caspase 3
Caspases