[The effects of hypoxia on the expression of inducible isoform of nitric oxide syntheses in nasal mucosal epithelial cells]

Lin Chuang Er Bi Yan Hou Ke Za Zhi. 2002 Aug;16(8):390-2.
[Article in Chinese]

Abstract

Objective: To study the synthesis of inducible isoform of nitric oxide synthase (iNOS) in human nasal mucosal epithelial cells (HNMECs) under hypoxia, and the role of dexamethasone on this process.

Method: HNMECs were cultured without serum under normal condition and hypoxia for 3-48 hours, parts of them were cultured with dexamethasone. Cell cycle of epithelial cells was observed by flowcytometer. INOS mRNA was detected by in situ hybridization.

Result: Cell cycle of epithelial cells became long under hypoxia. iNOS mRNA expression increased with time dependence after hypoxia for 3 h compared with controls. The peak appeared after 12 h hypoxia (P < 0.01). Increased iNOS mRNA expressions were reduced significantly by dexamethasone, but higher doses of dexamethasone (> 4.0 g/L) did not result in further suppression.

Conclusion: Hypoxia can regulate expression of iNOS mRNA significantly in HNMECs, and dexamethasone can restrain this process significantly.

MeSH terms

  • Anti-Inflammatory Agents / pharmacology
  • Cells, Cultured
  • Dexamethasone / pharmacology
  • Epithelial Cells / enzymology
  • Humans
  • Hypoxia / enzymology*
  • Nasal Mucosa / enzymology*
  • Nitric Oxide Synthase / biosynthesis*
  • Nitric Oxide Synthase / drug effects
  • Nitric Oxide Synthase / genetics
  • Nitric Oxide Synthase Type II
  • RNA, Messenger / biosynthesis
  • RNA, Messenger / drug effects

Substances

  • Anti-Inflammatory Agents
  • RNA, Messenger
  • Dexamethasone
  • NOS2 protein, human
  • Nitric Oxide Synthase
  • Nitric Oxide Synthase Type II