Stimulation of cortical acetylcholine release following blockade of ionotropic glutamate receptors in nucleus accumbens

Eur J Neurosci. 2002 Oct;16(7):1259-66. doi: 10.1046/j.1460-9568.2002.02201.x.

Abstract

In vivo microdialysis techniques were used to determine the ability of glutamate receptors within the nucleus accumbens to trans-synaptically modulate the basal forebrain cortical cholinergic system. Rats were implanted with a dialysis probe in the medial prefrontal cortex to measure changes in cortical acetylcholine efflux and in the ipsilateral nucleus accumbens to locally manipulate glutamate receptor activity. Intra-accumbens perfusion of the broad spectrum ionotropic glutamate receptor antagonist kynurentate (1.0, 5.0 mm) led to a dose-dependent increase (maximum of 200%) in cortical acetylcholine efflux. This stimulated efflux was reproduced with the intra-accumbens perfusion of the AMPA/kainate antagonist DNQX (0.1, 0.25, 2.5 mm; maximum increase of 200%) or the NMDA antagonist D-CPP (10.0, 100.0, 200 micro M; maximum increase of 400%). These results reveal a significant glutamatergic tone within the accumbens of awake rats and support the hypothesis that accumbens efferents to basal forebrain modulate the excitability of the basal forebrain cortical cholinergic system.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acetylcholine / analysis*
  • Animals
  • Dose-Response Relationship, Drug
  • Excitatory Amino Acid Antagonists / administration & dosage
  • Excitatory Amino Acid Antagonists / pharmacology*
  • Kynurenic Acid / administration & dosage
  • Kynurenic Acid / pharmacology*
  • Male
  • Microdialysis
  • N-Methylaspartate / antagonists & inhibitors
  • Nucleus Accumbens / metabolism
  • Prefrontal Cortex / drug effects
  • Prefrontal Cortex / metabolism*
  • Quinoxalines / pharmacology
  • Rats

Substances

  • Excitatory Amino Acid Antagonists
  • Quinoxalines
  • FG 9041
  • N-Methylaspartate
  • Kynurenic Acid
  • Acetylcholine