Nitric oxide in atherosclerosis

Mini Rev Med Chem. 2002 Dec;2(6):585-93. doi: 10.2174/1389557023405666.

Abstract

*NO is produced endogenously from L-arginine by NOSs. Among its multiple activities, the homeostatic control of the vascular endothelium is crucial for atherosclerosis, a pathogenic condition connected with elevated levels of LDL, the main plasma cholesterol carrier. Oxidised LDL is proatheromatic, and toxic peroxidation products contribute to further endothelial damage. *NO controls vascular tone, inhibits LDL oxidation and has hypocholesterolaemic activity. This review is referred to the chemistry, biology and role of *NO on atherosclerosis and its treatment by *NO donors or modulators.

Publication types

  • Review

MeSH terms

  • Arteriosclerosis / drug therapy
  • Arteriosclerosis / metabolism*
  • Cholesterol / metabolism
  • Humans
  • Lipid Peroxidation / drug effects
  • Lipid Peroxidation / physiology
  • Nitric Oxide / biosynthesis*
  • Nitric Oxide / chemistry
  • Nitric Oxide Donors / therapeutic use
  • Nitric Oxide Synthase / metabolism
  • Oxidation-Reduction
  • Reactive Oxygen Species / metabolism

Substances

  • Nitric Oxide Donors
  • Reactive Oxygen Species
  • Nitric Oxide
  • Cholesterol
  • Nitric Oxide Synthase