Fatty acids potentiate interleukin-1beta toxicity in the beta-cell line INS-1E

Biochem Biophys Res Commun. 2002 Aug 9;296(1):189-93. doi: 10.1016/s0006-291x(02)00819-7.

Abstract

Evidence for "lipotoxicity," i.e., negative effects of fatty acids (FA) on pancreatic beta-cells is incomplete. Here, we tested whether non-toxic concentrations of FA potentiate toxic effects of interleukin-1beta (IL-1beta). Culture of INS-1E clonal beta-cells for 2-6 days with 70 microM docosahexaenoic acid (DHA), eicosapentaenoic acid, arachidonic acid, 0.1mM linoleic acid, or 0.1-0.2mM oleic acid exerted no or minor negative effects on cell viability (MTT assay). Viability was reduced by 0.5 ng/ml IL-1beta. All tested FA significantly aggravated this effect after 6 days of culture. IL-1beta also exerted negative effects on cellular insulin content and DHA and oleic acid aggravated these effects. L-NAME, an inhibitor of constitutive nitric oxide (NO) synthase, reduced the negative effects of IL-1beta per se but did not abolish the potentiating effects of FA.

Conclusion: FA potentiate toxic effects of IL-1beta on beta-cells by mechanisms that include NO-independent ones.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Clone Cells
  • Drug Synergism
  • Fatty Acids / pharmacology*
  • Insulin / metabolism
  • Interleukin-1 / toxicity*
  • Islets of Langerhans / cytology
  • Islets of Langerhans / drug effects*
  • Islets of Langerhans / metabolism
  • NG-Nitroarginine Methyl Ester / pharmacology

Substances

  • Fatty Acids
  • Insulin
  • Interleukin-1
  • NG-Nitroarginine Methyl Ester