Abstract
Monocytes and macrophages play a pathogenic role in a number of autoimmune inflammatory diseases. Recent studies in experimental autoimmune encephalomyelitis (EAE), the animal model of multiple sclerosis, have identified a critical chemokine-mediated mechanism of monocyte homing to the central nervous system (CNS). Here, we summarize the current findings in EAE, develop a rationale for targeting the chemokine axis in order to treat CNS inflammatory disease, and review currently available molecule-specific therapeutics that inhibit monocyte trafficking to the CNS.
(c) 2002 Elsevier Science (USA).
Publication types
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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Animals
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Autoimmune Diseases / etiology
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Autoimmune Diseases / immunology*
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Autoimmune Diseases / therapy
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Central Nervous System Diseases / etiology
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Central Nervous System Diseases / immunology*
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Central Nervous System Diseases / therapy
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Chemokine CCL2 / metabolism
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Chemokines / metabolism
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Disease Models, Animal
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Encephalomyelitis, Autoimmune, Experimental / etiology
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Encephalomyelitis, Autoimmune, Experimental / immunology
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Humans
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Mice
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Mice, Knockout
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Models, Immunological
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Monocytes / immunology*
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Multiple Sclerosis / etiology
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Multiple Sclerosis / immunology
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Receptors, CCR2
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Receptors, Chemokine / metabolism
Substances
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CCR2 protein, human
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Ccr2 protein, mouse
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Chemokine CCL2
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Chemokines
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Receptors, CCR2
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Receptors, Chemokine