Rescue of trophoblast apoptosis by parathyroid hormone-related protein

BJOG. 2002 Feb;109(2):218-20. doi: 10.1111/j.1471-0528.2002.01033.x.

Abstract

The effects of the 1-34 N-terminal parathyroid hormone-related protein fragment (0-10 microM) and the 67-86 parathyroid hormone-related protein fragment (0-10 microM) on trophoblast apoptosis, induced by TNFalpha and IFN-gamma or by the protein kinase inhibitor staurosporine, were investigated. TNFalpha/IFN-gamma and staurosporine significantly increased the rate of apoptosis by fivefold and by eightfold, respectively. Parathyroid hormone-related protein (1-34) evoked a dose-dependent rescue of both TNFalpha/IFNgamma-induced and staurosporine-induced apoptosis, whereas parathyroid hormone-related protein (67-86) had no significant effect on staurosporine-induced apoptosis, and only significantly diminished TNFalpha/IFNgamma-induced apoptosis at 10 microM. Parathyroid hormone-related protein was thus found to be a cytotrophoblast apoptosis survival factor.

MeSH terms

  • Adult
  • Apoptosis / drug effects
  • Apoptosis / physiology*
  • Dose-Response Relationship, Drug
  • Enzyme Inhibitors / pharmacology
  • Female
  • Gestational Age
  • Humans
  • Interferon-gamma / pharmacology
  • Parathyroid Hormone-Related Protein
  • Pregnancy / physiology*
  • Proteins / physiology*
  • Staurosporine / pharmacology
  • Trophoblasts / cytology
  • Trophoblasts / drug effects*
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • Enzyme Inhibitors
  • PTHLH protein, human
  • Parathyroid Hormone-Related Protein
  • Proteins
  • Tumor Necrosis Factor-alpha
  • Interferon-gamma
  • Staurosporine