Treatment with anti-CD154 antibody and donor-specific transfusion prevents acute rejection of myoblast transplantation

Transplantation. 2002 Feb 15;73(3):453-61. doi: 10.1097/00007890-200202150-00021.

Abstract

Background: Achieving immunological tolerance to transplanted myoblasts would reduce the adverse effects associated with the sustained immunosuppression required for this experimental therapeutic approach in Duchenne muscular dystrophic patients.

Methods: Mdx mice were transplanted with fully allogeneic BALB/c myoblasts in the tibialis anterior muscles. Seven days before transplantation (-7), host mice received 107 total donor spleen cells i.v. (donor-specific transfusion, DST) with 500 microg of anti-CD154 mAb i.p. on days -7, -4, 0, +4.

Results: Results showed a high level of dystrophin expression in 83, 60, and 20% of the mice 1, 3, and 6 months, respectively, after transplantation of myoblasts. No antibodies against the donor cells were produced up to 3 months after transplantation. However, abundant activated cytotoxic cells were present in muscles still expressing high percentage of dystrophin positive fibers.

Conclusions: In conclusion, the DST + anti-CD154 mAb treatments effectively prolonged myoblast survival, but this treatment could not develop tolerance to complete allogeneic myoblast transplantation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Disease
  • Animals
  • Antibodies, Monoclonal / therapeutic use*
  • Blood Transfusion*
  • CD4-Positive T-Lymphocytes / physiology
  • CD40 Ligand / immunology*
  • CD40 Ligand / physiology
  • CD8-Positive T-Lymphocytes / physiology
  • Cell Transplantation*
  • Cytokines / genetics
  • Dystrophin / analysis
  • Graft Rejection / prevention & control*
  • Granzymes
  • Mice
  • Mice, Inbred BALB C
  • Muscles / chemistry
  • Muscles / cytology*
  • Muscular Dystrophies / metabolism
  • Muscular Dystrophies / therapy*
  • RNA, Messenger / analysis
  • Serine Endopeptidases / genetics

Substances

  • Antibodies, Monoclonal
  • Cytokines
  • Dystrophin
  • RNA, Messenger
  • CD40 Ligand
  • Granzymes
  • Gzmb protein, mouse
  • Serine Endopeptidases