Clinical relevance of precore and basal core promoter variants of hepatitis B virus during natural hepatitis B e antigen seroconversion may be overstated

J Pediatr Gastroenterol Nutr. 2001 Sep;33(3):301-6. doi: 10.1097/00005176-200109000-00014.

Abstract

Background: Clinical relevance of nucleotide changes in precore and basal core promoters in the hepatitis B virus genome during hepatitis B e antigen seroconversion may be overstated. The authors investigated the existence and changes in the relative proportion of variants to wild virus that occur with seroconversion.

Methods: Sera from 30 school-aged long-term hepatitis B virus carriers, including 11 tested before and after seroconversion during 1 to 8 years of follow-up, were evaluated for variations in nucleotide sequences of the basal core promoter (T1762 and A1764), precore region (A1869), and carboxyl-terminus of the X region of the hepatitis B virus genome using an amplification refractory mutation detection system with mutant-specific primers.

Results: All variants were found to already exist before seroconversion at various wild-type/mutant ratios. The positive rates of these variants were not changed with loss of hepatitis B e antigen. Although there was a relative increase in the concentration of these mutants in wild-type/mutant mixed populations, most patients with only a wild-type population maintained the same pattern after loss of hepatitis B e antigen.

Conclusions: Our results indicate that hepatitis B virus exists as a quasi species, and correlations of nucleotide sequences with clinical and serologic findings must be done with caution.

MeSH terms

  • Adolescent
  • Child
  • Cross-Sectional Studies
  • DNA Primers
  • Female
  • Follow-Up Studies
  • Gene Amplification
  • Genetic Variation
  • Genome, Viral
  • Hepatitis B / immunology
  • Hepatitis B / virology*
  • Hepatitis B e Antigens / blood*
  • Hepatitis B virus / genetics*
  • Hepatitis B virus / immunology
  • Humans
  • Male
  • Mutation
  • Polymerase Chain Reaction
  • Prevalence
  • Promoter Regions, Genetic
  • Sensitivity and Specificity

Substances

  • DNA Primers
  • Hepatitis B e Antigens