Th1 and Th2 cytokine profiles in recurrent aborters with successful pregnancy and with subsequent abortions

Hum Reprod. 2001 Oct;16(10):2219-26. doi: 10.1093/humrep/16.10.2219.

Abstract

Background: This study compared Th1-Th2 cytokine profiles in a subgroup of recurrent aborters who had an abortion with those in a subgroup of recurrent aborters who had a successful pregnancy.

Methods: Fifty-four women with a history of at least three normal pregnancies, 24 women with a history of recurrent spontaneous abortion (RSA) followed by abortion (RSA-->A) and 39 women with a history of RSA followed by normal pregnancy (RSA-->N) were studied. Blood samples and placentas were obtained at the time of delivery or abortion; peripheral blood mononuclear cells were stimulated separately with phytohaemagglutinin and with autologous placental cells, and the secreted cytokines estimated.

Results: Peripheral blood mononuclear cells from the RSA-->N subgroup secreted higher concentrations of Th1-type cytokines as compared with normal pregnant women, indicating a higher Th1 bias in these women. However, women in the RSA-->N subgroup had significantly higher concentrations of Th2 cytokines as compared with women in the RSA-->A subgroup. A comparison of Th1:Th2 cytokine ratios indicated a higher Th2 bias in RSA-->N women as compared with RSA-->A women.

Conclusions: We conclude that abortion-prone women who proceed to have successful pregnancy are more Th2-biased than abortion-prone women who abort, and that recurrent aborters who undergo spontaneous abortion have a stronger Th1 bias than aborters who have normal pregnancy.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Abortion, Habitual / blood*
  • Cell Division
  • Cells, Cultured
  • Coculture Techniques
  • Cytokines / blood*
  • Female
  • Humans
  • Lymphocyte Culture Test, Mixed
  • Mitogens / pharmacology
  • Monocytes / immunology
  • Monocytes / pathology
  • Placenta / immunology
  • Placenta / pathology
  • Pregnancy / blood*
  • Th1 Cells / drug effects
  • Th1 Cells / metabolism*
  • Th2 Cells / drug effects
  • Th2 Cells / metabolism*

Substances

  • Cytokines
  • Mitogens