Src-catalyzed phosphorylation of c-Cbl leads to the interdependent ubiquitination of both proteins

J Biol Chem. 2001 Sep 14;276(37):35185-93. doi: 10.1074/jbc.M102219200. Epub 2001 Jul 11.

Abstract

The protooncogene c-Cbl has recently emerged as an E3 ubiquitin ligase for activated receptor tyrosine kinases. We report here that c-Cbl also mediates the ubiquitination of another protooncogene, the non-receptor tyrosine kinase c-Src, as well as of itself. The c-Cbl-dependent ubiquitination of Src and c-Cbl requires c-Cbl's RING finger, Src kinase activity, and c-Cbl's tyrosine phosphorylation, probably on Tyr-371. In vitro, c-Cbl forms a stable complex with the ubiquitin-conjugating enzyme UbcH7, but active Src destabilizes this interaction. In contrast, Src inhibition stabilizes the c-Cbl. UbcH7.Src complex. Finally, c-Cbl reduces v-Src protein levels and suppresses v-Src-induced STAT3 activation. Thus, in addition to mediating the ubiquitination of activated receptor tyrosine kinases, c-Cbl also acts as a ubiquitin ligase for the non-receptor tyrosine kinase Src, thereby down-regulating Src.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Binding Sites
  • DNA-Binding Proteins / antagonists & inhibitors
  • Gene Expression Regulation
  • Oncogene Protein pp60(v-src) / analysis
  • Phosphorylation
  • Proto-Oncogene Proteins / physiology*
  • Proto-Oncogene Proteins c-cbl
  • STAT3 Transcription Factor
  • Trans-Activators / antagonists & inhibitors
  • Ubiquitin-Protein Ligases*
  • Ubiquitins / metabolism*
  • src-Family Kinases / physiology*

Substances

  • DNA-Binding Proteins
  • Proto-Oncogene Proteins
  • STAT3 Transcription Factor
  • Trans-Activators
  • Ubiquitins
  • Proto-Oncogene Proteins c-cbl
  • Ubiquitin-Protein Ligases
  • Oncogene Protein pp60(v-src)
  • src-Family Kinases