Abstract
Hereditary nonpolyposis colorectal cancer (HNPCC) is a common inherited form of neoplasia caused by germline mutations in DNA mismatch repair (MMR) genes. MMR proteins have been reported to associate with several proteins, including the human exonuclease 1 (hEXO1). We report here novel HNPCC-hMLH1 mutant proteins (T117M, Q426X and 1813insA) in Danish HNPCC patients. We demonstrate that these mutant HNPCC-hMLH1 proteins are unable to form complexes with hEXO1 and hPMS2 in vivo. The results indicate that mutations found in HNPCC gene carriers disrupt hMLH1-hEXO1 complex formation and hMutLalpha heterodimer assembly essential for MMR activity.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Adaptor Proteins, Signal Transducing
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Adenosine Triphosphatases*
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Amino Acid Sequence
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Amino Acid Substitution
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Bacterial Proteins / chemistry
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Bacterial Proteins / metabolism*
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Base Pair Mismatch*
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Carrier Proteins
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Colorectal Neoplasms, Hereditary Nonpolyposis / genetics*
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DNA Repair Enzymes*
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DNA Repair*
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DNA Transposable Elements
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DNA-Binding Proteins*
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Denmark
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Escherichia coli
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Escherichia coli Proteins*
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Exodeoxyribonucleases / metabolism*
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Humans
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Mismatch Repair Endonuclease PMS2
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Molecular Sequence Data
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MutL Protein Homolog 1
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MutL Proteins
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Mutation*
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Mutation, Missense
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Neoplasm Proteins / chemistry
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Neoplasm Proteins / genetics*
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Neoplasm Proteins / metabolism*
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Nuclear Proteins
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Recombinant Fusion Proteins / chemistry
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Recombinant Fusion Proteins / metabolism
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Sequence Alignment
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Sequence Homology, Amino Acid
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White People
Substances
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Adaptor Proteins, Signal Transducing
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Bacterial Proteins
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Carrier Proteins
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DNA Transposable Elements
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DNA-Binding Proteins
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Escherichia coli Proteins
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MLH1 protein, human
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MutL protein, E coli
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Neoplasm Proteins
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Nuclear Proteins
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Recombinant Fusion Proteins
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EXO1 protein, human
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Exodeoxyribonucleases
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exodeoxyribonuclease I
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Adenosine Triphosphatases
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PMS2 protein, human
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Mismatch Repair Endonuclease PMS2
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MutL Protein Homolog 1
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MutL Proteins
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DNA Repair Enzymes