Increasing specificity of anti-tumor therapy: cytotoxic protein delivery by non-pathogenic clostridia under regulation of radio-induced promoters

Anticancer Res. 2001 Mar-Apr;21(2A):857-61.

Abstract

Background: Pathogenic clostridia, genetically engineered to express therapeutic genes, will specifically target hypoxic regions in tumors. This specificity can be further improved if expression of these genes is controlled by a radio-induced promoter, leading to spatial and temporal control of gene expression.

Materials and methods: Following administration of Clostridium spores to tumor bearing rats, normal tissue and tumoral specimens were compared for colonization. Clostridium was genetically modified to express tumor necrosis factor a or cytosine deaminase. Expression of these proteins was assayed. Northern blot hybridizations were used to detect genes which are radio-induced.

Results: Clostridium gave a selective colonization of tumors. The recombinant clostridia expressed in vitro and in vivo TNF alpha and cytosine deaminase. Clostridial SOS-repair genes were induced at a dose of 2 Gy.

Conclusions: Pathogenic Clostridium can be used for tumor specific delivery of therapeutic genes. The specificity can be improved via radio-induced promoters. Overall, this new gene delivery system can lead to an increase of the therapeutic ratio in cancer treatment.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antineoplastic Agents / administration & dosage*
  • Bacterial Proteins / genetics
  • Cellulase / genetics*
  • Clostridium* / genetics
  • Cytosine Deaminase
  • DNA Restriction Enzymes*
  • Deoxyribonucleases / genetics
  • Drug Delivery Systems
  • Flucytosine
  • Fluorouracil
  • Genetic Therapy
  • Genetic Vectors* / genetics
  • Male
  • Nucleoside Deaminases / administration & dosage
  • Nucleoside Deaminases / genetics*
  • Nucleoside Deaminases / therapeutic use
  • Promoter Regions, Genetic*
  • Rats
  • Rec A Recombinases / genetics
  • Tumor Necrosis Factor-alpha / administration & dosage
  • Tumor Necrosis Factor-alpha / genetics*
  • Tumor Necrosis Factor-alpha / therapeutic use

Substances

  • Antineoplastic Agents
  • Bacterial Proteins
  • Tumor Necrosis Factor-alpha
  • Flucytosine
  • Rec A Recombinases
  • Deoxyribonucleases
  • DNA Restriction Enzymes
  • RecN protein, Bacteria
  • Cellulase
  • Nucleoside Deaminases
  • Cytosine Deaminase
  • Fluorouracil