Cytokine regulation of gap junction connectivity: an open-and-shut case or changing partners at the Nexus?

Am J Pathol. 2001 May;158(5):1565-9. doi: 10.1016/S0002-9440(10)64110-7.

Authors

C F Brosnan¹, E Scemes, D C Spray

Affiliation

¹ Department of Pathology, Albert Einstein College of Medicine, Bronx, New York, USA. brosnan@aecom.yu.edu

No abstract available

Publication types

Comment

MeSH terms

Bronchi / cytology
Bronchi / drug effects*
Bronchi / metabolism
Cell Communication / drug effects
Connexins / genetics
Cystic Fibrosis / physiopathology
Cystic Fibrosis Transmembrane Conductance Regulator / genetics
Cystic Fibrosis Transmembrane Conductance Regulator / physiology
Cytokines / pharmacology*
Fluorescent Dyes / metabolism
Gap Junctions / drug effects*
Gene Expression Regulation / drug effects
Humans
Isoquinolines / metabolism
RNA, Messenger / drug effects
RNA, Messenger / genetics
RNA, Messenger / metabolism
Tumor Necrosis Factor-alpha / pharmacology

Substances

CFTR protein, human
Connexins
Cytokines
Fluorescent Dyes
Isoquinolines
RNA, Messenger
Tumor Necrosis Factor-alpha
Cystic Fibrosis Transmembrane Conductance Regulator
lucifer yellow