Molecular scanning for mutations in the melanocortin-4 receptor gene in obese/diabetic Japanese

Ann Hum Genet. 1999 Nov;63(Pt 6):483-7. doi: 10.1017/S0003480099007782.

Abstract

Decreased function of the melanocortin-4 receptor (MC4R) was reported to cause late-onset obesity and insulin resistance in rodents. Thus mutations in the MC4R gene drew strong attention as a possible cause of obesity and diabetes. We screened for mutations in the MC4R gene in extremely obese [body mass index (BMI) > or = 35 kg/m2] Japanese with diabetes by direct sequencing. A heterozygous mutation (V103I) was detected in one case (2.0 %), however the frequency was not significantly different from that in non-obese (BMI < or = 24 kg/m2) and non-diabetic subjects (2.7 %). No other mutations were detected. These results suggest that mutations including V103I in the MC4R gene are not a major cause of obesity or diabetes in Japanese.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • Case-Control Studies
  • Codon / genetics
  • DNA Mutational Analysis
  • DNA Primers / genetics
  • Diabetes Mellitus, Type 2 / complications*
  • Diabetes Mellitus, Type 2 / genetics*
  • Female
  • Genotype
  • Humans
  • Insulin Resistance / genetics
  • Japan
  • Male
  • Middle Aged
  • Mutation*
  • Obesity, Morbid / complications*
  • Obesity, Morbid / genetics*
  • Receptor, Melanocortin, Type 4
  • Receptors, Corticotropin / genetics*

Substances

  • Codon
  • DNA Primers
  • Receptor, Melanocortin, Type 4
  • Receptors, Corticotropin