CRE-decoy oligonucleotide-inhibition of gene expression and tumor growth

Mol Cell Biochem. 2000 Sep;212(1-2):29-34.

Abstract

Nucleic acid molecules with high affinities for a target transcription factor can be introduced into cells as decoy cis-elements to bind these factors and alter gene expression. This review discusses a synthetic single-stranded palindromic oligonucleotide, which self-hybridizes to form a duplex/hairpin and competes with cAMP response element (CRE) enhancers for binding transcription factors. This oligonucleotide inhibits CRE- and Ap-1-directed gene transcription and promotes growth inhibition in vitro and in vivo in a broad spectrum of cancer cells, without adversely affecting normal cell growth. Evidence presented here suggests that the CRE-decoy oligonucleotide can provide a powerful new means of combating cancers, viral diseases, and other pathological conditions by regulating the expression of cAMP-responsive genes.

Publication types

  • Review

MeSH terms

  • Animals
  • Cell Division / drug effects*
  • Cyclic AMP Response Element-Binding Protein / metabolism*
  • Enhancer Elements, Genetic
  • Gene Expression Regulation / drug effects*
  • Genes, fos
  • Humans
  • Oligodeoxyribonucleotides / pharmacology*
  • Oligodeoxyribonucleotides / therapeutic use
  • Transcription Factor AP-1 / metabolism
  • Transcription, Genetic / drug effects*
  • Tumor Cells, Cultured
  • Tumor Suppressor Protein p53 / metabolism

Substances

  • Cyclic AMP Response Element-Binding Protein
  • Oligodeoxyribonucleotides
  • Transcription Factor AP-1
  • Tumor Suppressor Protein p53