A role for the endogenous cannabinoid system in the peripheral control of pain initiation

Prog Brain Res. 2000:129:471-82. doi: 10.1016/s0079-6123(00)29034-1.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Amides
  • Analgesics / pharmacology
  • Animals
  • Arachidonic Acids / biosynthesis
  • Arachidonic Acids / pharmacology
  • Brain / cytology
  • Brain / drug effects
  • Brain / metabolism
  • Cannabinoids / biosynthesis*
  • Drug Interactions / physiology
  • Endocannabinoids
  • Ethanolamines
  • Ganglia, Spinal / cytology
  • Ganglia, Spinal / drug effects
  • Ganglia, Spinal / metabolism
  • Glycerides / biosynthesis
  • Humans
  • Nociceptors / cytology
  • Nociceptors / drug effects*
  • Nociceptors / metabolism
  • Pain / drug therapy*
  • Pain / pathology
  • Pain / physiopathology*
  • Palmitic Acids / pharmacology
  • Polyunsaturated Alkamides
  • Receptors, Cannabinoid
  • Receptors, Drug / drug effects
  • Receptors, Drug / metabolism
  • Signal Transduction / drug effects
  • Signal Transduction / physiology

Substances

  • Amides
  • Analgesics
  • Arachidonic Acids
  • Cannabinoids
  • Endocannabinoids
  • Ethanolamines
  • Glycerides
  • Palmitic Acids
  • Polyunsaturated Alkamides
  • Receptors, Cannabinoid
  • Receptors, Drug
  • palmidrol
  • glyceryl 2-arachidonate
  • anandamide