Abstract
Following transvenous implantable cardioverter defibrillator shocks, a significant increase in QT dispersion was observed. We suggest shock-induced increased dispersion of myocardial repolarization as one of the mechanisms of shock-induced proarrhythmia.
MeSH terms
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Cardiac Pacing, Artificial / methods*
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Defibrillators, Implantable* / classification
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Electrocardiography*
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Heart Rate / physiology
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Humans
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Monitoring, Physiologic
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Myocardial Contraction / physiology*
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Time Factors
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Ventricular Fibrillation / etiology
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Ventricular Fibrillation / physiopathology*
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Ventricular Fibrillation / therapy*