HCV-core protein accelerates recovery from the insensitivity of liver cells to Fas-mediated apoptosis induced by an injection of anti-Fas antibody in mice

J Hepatol. 2000 Sep;33(3):440-7. doi: 10.1016/s0168-8278(00)80280-9.

Abstract

Background/aims: Hepatitis C virus (HCV) is a major etiologic agent of chronic hepatitis, cirrhosis, and hepatocellular carcinoma. The aim of this study was to elucidate pathological effects of HCV-core protein on liver cells.

Methods: We have generated transgenic mice carrying HCV-core cDNA (Px-core) and pathologically examined livers of Px-core mice.

Results: HCV-core protein was detectable in livers from lines 5 (C5) and 8 (C8) of Px-core transgenic mice. Since chronic hepatitis and cirrhosis precede hepatocellular carcinoma in patients with HCV infection, we tried to examine the effect of repetitive injection of a small dose of anti-Fas antibody in the transgenic mice. Surprisingly, an initial injection of anti-Fas antibody induced resistance of liver cells to the second injection of anti-Fas antibody in both Px-core and littermate control mice. The insensitivity of liver cells induced in the control mice continued for more than 24 weeks after the first injection but was broken within 1 week after partial hepatectomy. However, the sensitivity was restored in the Px-core mice within 12 weeks after the injection.

Conclusion: HCV-core protein in liver cells may affect persistence of Fas-mediated liver cell injury.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies / pharmacology*
  • Apoptosis / drug effects*
  • Drug Resistance
  • Gene Expression
  • Liver / pathology
  • Liver / physiopathology*
  • Mice
  • Mice, Inbred Strains
  • Mice, Transgenic / genetics
  • Time Factors
  • Viral Core Proteins / genetics
  • Viral Core Proteins / pharmacology*
  • fas Receptor / immunology*
  • fas Receptor / physiology*

Substances

  • Antibodies
  • Viral Core Proteins
  • fas Receptor
  • nucleocapsid protein, Hepatitis C virus