Phloretin inhibits Na+ and K+ uptake in cultured alveolar type II cells by reduction of cellular ATP content

Exp Lung Res. 2000 Jul-Aug;26(5):319-33. doi: 10.1080/019021400408281.

Abstract

A number of Na+ and K+ transport pathways have been identified in the alveolar epithelium and multiple inhibitors have been used to uncover these mechanisms. However, the effect of phloretin, a small dipolar nonelectrolyte compound which exerts many effects on membrane transport on Na+ and K+ uptake in alveolar epithelial cells, is not known. The purpose of this study was then to determine the impact of phloretin in Na+ and K+ uptake in cultured rat alveolar type II cells. Phloretin at a dose of 250 microM decreased Na+ uptake by 80% and K+ uptake by 90%. This decrease in Na+ and K+ uptake was not associated with a cytotoxic effect of phloretin, but this treatment did decrease ATP levels in the cells to 80% of the control cells value by 5 minutes and to 95% by 10 minutes. Our study demonstrates that phloretin is a nonspecific inhibitor of ions transport in alveolar type II cells. This inhibition is probably mediated by a reduction of intracellular ATP content.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Triphosphate / metabolism*
  • Animals
  • Cells, Cultured
  • Dose-Response Relationship, Drug
  • Epithelium / drug effects
  • Epithelium / metabolism
  • Ion Transport / drug effects
  • L-Lactate Dehydrogenase / metabolism
  • Male
  • Phloretin / pharmacology*
  • Potassium / metabolism*
  • Pulmonary Alveoli / cytology
  • Pulmonary Alveoli / drug effects
  • Pulmonary Alveoli / metabolism*
  • Rats
  • Rats, Sprague-Dawley
  • Sodium / metabolism*

Substances

  • Adenosine Triphosphate
  • Sodium
  • L-Lactate Dehydrogenase
  • Potassium
  • Phloretin