Effect of acute L-Dopa pretreatment on apomorphine-induced rotational behavior in a rat model of Parkinson's disease

Exp Neurol. 2000 Jan;161(1):212-9. doi: 10.1006/exnr.1999.7245.

Abstract

Currently, reduction of apomorphine-induced rotational behavior in the 6-hydroxydopamine (6-OHDA) lesioned rat is the most utilized drug-induced paradigm for assessing functional efficacy in a rat model of Parkinson's disease (PD). Any clinically predictive animal model of PD should include a positive response to l-dopa, the standard pharmacotherapy for PD. However, the acute interaction between L-dopa and apomorphine has never been studied to determine if L-dopa pretreatment could reduce apomorphine-induced rotational behavior in a 6-OHDA lesioned rat. The present study was designed to explore whether, indeed, pretreatment with subrotational doses of L-dopa could inhibit apomorphine-induced rotations. The data indicate that L-dopa significantly reduced apomorphine-induced rotational behavior only at one dose (5.0 mg/kg) for 12 min. Based on these and other data, it is concluded that although the apomorphine-induced rotational paradigm may continue to be utilized as one additional indicator of efficacy in the 6-OHDA rat model of PD, it is not in itself a completely valid functional assay.

MeSH terms

  • Animals
  • Antiparkinson Agents / pharmacology*
  • Apomorphine / pharmacology*
  • Behavior, Animal / drug effects*
  • Carbidopa / pharmacology
  • Corpus Striatum / physiology
  • Disease Models, Animal
  • Dopamine / physiology
  • Dopamine Agents / pharmacology
  • Dose-Response Relationship, Drug
  • Levodopa / pharmacology*
  • Male
  • Oxidopamine
  • Parkinson Disease, Secondary / drug therapy*
  • Parkinson Disease, Secondary / physiopathology
  • Rats
  • Rats, Inbred F344
  • Rotation
  • Substantia Nigra / physiology
  • Sympatholytics

Substances

  • Antiparkinson Agents
  • Dopamine Agents
  • Sympatholytics
  • Levodopa
  • Oxidopamine
  • Carbidopa
  • Apomorphine
  • Dopamine