Dominant T cells in idiopathic nephrotic syndrome of childhood

Kidney Int. 2000 Feb;57(2):510-7. doi: 10.1046/j.1523-1755.2000.00870.x.

Abstract

Background: Because of several studies, idiopathic nephrotic syndrome (INS) of childhood is suspected to have an immunologic pathogenesis with T cells playing a major role. To investigate this hypothesis further, we studied the diversity of the CDR3 region of the T-cell receptor (TCR) beta-chain from peripheral T cells isolated from patients with INS.

Methods: The study was performed over a three-year period to obtain longitudinal data on the repertoire of peripheral T cells. mRNA from peripheral mononuclear cells (PBMCs) of seven INS patients and two healthy controls (NHD) was prepared and analyzed for CDR3 length polymorphism of TCR beta-chain by spectratyping.

Results: All INS patients presented individually skewed spectratype histograms in at least one Vbeta-family. Patients suffering from a frequent relapsing course of INS or a focal global sclerosis showed some alterations to persist in all samples isolated in the observation period (up to 3 years). In addition, sequence analyses of the beta-chain of the TCR CDR3 region confirmed clonal expansion of peripheral T cells in those patients who had displayed spectratype alterations.

Conclusions: The data give strong evidence for an direct involvement of CD8+ T cells in the complicated course of INS.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age of Onset
  • Amino Acid Sequence
  • CD4-Positive T-Lymphocytes / chemistry
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / chemistry
  • CD8-Positive T-Lymphocytes / immunology*
  • Child
  • Complementarity Determining Regions*
  • Gene Expression / immunology
  • Genes, T-Cell Receptor beta / genetics*
  • Genes, T-Cell Receptor beta / immunology
  • Humans
  • Immunoglobulin Variable Region / genetics
  • Molecular Sequence Data
  • Nephrotic Syndrome / etiology
  • Nephrotic Syndrome / immunology*
  • Nephrotic Syndrome / physiopathology
  • Polymorphism, Genetic
  • Receptors, Antigen, T-Cell, alpha-beta / chemistry
  • Receptors, Antigen, T-Cell, alpha-beta / genetics
  • Receptors, Antigen, T-Cell, alpha-beta / immunology
  • Sequence Analysis, DNA
  • T-Lymphocyte Subsets / immunology

Substances

  • Complementarity Determining Regions
  • Immunoglobulin Variable Region
  • Receptors, Antigen, T-Cell, alpha-beta