Effects of hemoglobin-based oxygen-carrying solutions in anesthetized rats with acute ischemic renal failure

J Lab Clin Med. 2000 Jan;135(1):73-81. doi: 10.1016/s0022-2143(00)70023-0.

Abstract

The effects of three hemoglobin solutions were compared with those of iso-oncotic human serum albumin in rats with ischemic renal failure and sham-operated controls. Unmodified and alpha-alpha cross-linked hemoglobins both increase mean arterial pressure and systemic vascular resistance and reduce cardiac output substantially and to a comparable extent. In contrast, omicron-raffinose cross-linked hemoglobin has no deleterious effect on any of these parameters. In sham-operated rats unmodified hemoglobin reduces the glomerular filtration rate (GFR) by approximately 30%, whereas neither of the two cross-linked hemoglobins has any adverse effect on GFR in this group. None of the three hemoglobin solutions exacerbated the degree to which GFR was reduced by ischemia-reperfusion injury. Also, the degree of tubular necrosis induced by ischemia-reperfusion injury was also comparable in all groups. We conclude the following: (1) omicron-raffinose cross-linking, but not alpha-alpha cross-linking, ameliorates the effects of unmodified hemoglobin on vascular resistance and cardiac output; (2) both forms of cross-linking reduce the nephrotoxicity exhibited by unmodified hemoglobin in sham-operated rats; and (3) none of the hemoglobin solutions exacerbate renal injury induced by ischemia-reperfusion.

Publication types

  • Comparative Study
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acute Kidney Injury / metabolism
  • Acute Kidney Injury / pathology
  • Acute Kidney Injury / physiopathology*
  • Animals
  • Aspirin / analogs & derivatives*
  • Aspirin / pharmacology
  • Blood Pressure / drug effects*
  • Glomerular Filtration Rate / drug effects
  • Heart Rate / drug effects
  • Hemoglobins / pharmacology*
  • Humans
  • Kidney Tubules / drug effects
  • Kidney Tubules / pathology
  • Kidney Tubules / physiopathology
  • Male
  • Necrosis
  • Oxygen / metabolism*
  • Raffinose / analogs & derivatives*
  • Raffinose / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Reperfusion Injury / metabolism
  • Reperfusion Injury / pathology
  • Reperfusion Injury / physiopathology
  • Serum Albumin / pharmacology
  • Sodium / urine
  • Vasoconstriction / drug effects*

Substances

  • Hemoglobins
  • O-raffinose cross-linked human hemoglobin
  • Serum Albumin
  • hemoglobin XL99alpha
  • Sodium
  • Raffinose
  • Aspirin
  • Oxygen