Aminoglycosides activate oxygen metabolites production in the cochlea of mature and developing rats

Hear Res. 1999 Oct;136(1-2):165-8. doi: 10.1016/s0378-5955(99)00122-7.

Abstract

The ototoxicity of antibiotics, particularly of aminoglycosides, is a well-known undesirable side effect which may be based on a free radical mechanism. We studied the effect of different antibiotics in the production of reactive oxygen species in freshly isolated cochleas of mature and 2-10 weeks old developing rats. Phorbol myristate acetate induced the release of reactive oxygen species after a lag time close to 30 s and declined back to basal values in 10-20 min. The rate of reactive oxygen species production correlated inversely to the age in 2-10 weeks old rats. The study of a set of antibiotics showed that a very low concentration of gentamicin and streptomycin (10-100 ng/ml) enhanced the effect of phorbol myristate acetate. At the above-indicated concentrations, ciprofloxacin did not modify phorbol myristate acetate-induced activation. These results show the enhancement by aminoglycosides of reactive oxygen species production in cochlear tissues, particularly in developing rats.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging / metabolism*
  • Animals
  • Animals, Newborn / growth & development
  • Animals, Newborn / metabolism*
  • Anti-Bacterial Agents / pharmacology*
  • Anti-Infective Agents / pharmacology
  • Ciprofloxacin / pharmacology
  • Cochlea / drug effects
  • Cochlea / metabolism*
  • Drug Synergism
  • Gentamicins / pharmacology
  • In Vitro Techniques
  • Rats
  • Rats, Wistar
  • Reactive Oxygen Species / metabolism*
  • Streptomycin / pharmacology
  • Tetradecanoylphorbol Acetate / pharmacology

Substances

  • Anti-Bacterial Agents
  • Anti-Infective Agents
  • Gentamicins
  • Reactive Oxygen Species
  • Ciprofloxacin
  • Tetradecanoylphorbol Acetate
  • Streptomycin