Dilatation and constriction of rat gastric mucosal microvessels through prostaglandin EP2 and EP3 receptors

Aliment Pharmacol Ther. 1999 Sep;13(9):1243-50. doi: 10.1046/j.1365-2036.1999.00577.x.

Abstract

Background: Prostaglandin (PG)E2 has both a vasodilating action and a protective function in the gastric mucosa. There are four subtypes of PGE2-sensitive, or EP, receptors.

Aim: To identify the subtype of EP receptors in the microvessels of the rat gastric mucosa using EP2 and EP3 receptor agonists.

Methods: The posterior wall of the anaesthetized rat stomach was secured in a chamber and superfused with Tyrode's solution, and the gastric microcirculation of the mucosal base was observed through a window with transillumination. PGE2 and its derivatives (20 microL) were applied topically in the window.

Results: PGE2 (0.001-10 micromol/L), misoprostol (EP2/EP3 receptor agonist; 0.01-100 micromol/L) and butaprost (EP2 receptor agonist; 1-1000 micromol/L) dilated the arterioles dose-dependently, but M&B 28 767 (EP3 receptor agonist; 0.001-10 micromol/L) did not alter their diameters. M&B 28 767 constricted the venules and collecting venules dose-dependently whereas butaprost dilated them. PGE2 and misoprostol had bell-shaped dose-response curves: constriction by low doses of PGE2 and misoprostol (0.001-0.1 micromol/L and 0.01-1 micromol/L) and dilation by high doses of PGE2 and misoprostol (0.1-100 micromol/L and 1-100 micromol/L).

Conclusions: These results suggest that PGE2 dilated both arterioles and venules in the rat gastric mucosa through the EP2 receptors and constricted the venules through the EP3 receptors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alprostadil / analogs & derivatives*
  • Alprostadil / pharmacology
  • Analysis of Variance
  • Animals
  • Dinoprostone / pharmacology*
  • Gastric Mucosa / blood supply
  • Gastric Mucosa / drug effects*
  • Male
  • Microcirculation
  • Misoprostol / pharmacology*
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Prostaglandin E / agonists*
  • Receptors, Prostaglandin E / antagonists & inhibitors*
  • Vasoconstriction / drug effects
  • Vasoconstriction / physiology
  • Vasodilation / drug effects
  • Vasodilation / physiology

Substances

  • Receptors, Prostaglandin E
  • Misoprostol
  • 11-deoxy-16-phenoxy-17,18,19,20-tetranorprostaglandin E1
  • Alprostadil
  • butaprost
  • Dinoprostone