cAMP-independent chloride secretion activated by a vasoactive intestinal peptide in a monolayer culture of human bronchial epithelial cells

Biol Pharm Bull. 1999 Jul;22(7):745-8. doi: 10.1248/bpb.22.745.

Abstract

To investigate the effects of vasoactive intestinal peptide (VIP) on Cl- transport across normal human bronchial epithelial (NHBE) cells grown in a monolayer, changes in short-circuit current (Isc) were measured in Ussing chamber systems. In the presence of 10(-4) M amiloride, the addition of VIP to the serosal solution led to an increase in the Isc in a concentration-dependent manner, the 50% effective concentration (EC50) being 2.6 x 10(-11) M. However, the addition of 10(-5) M forskolin had little effect on the increase in Isc. On the other hand, in the intracellular cAMP measurement, 10(-5) M forskolin remarkably increased the cAMP levels, but 10(-7) M VIP did not. This result suggests that Cl- secretion by VIP is not related to the raised intracellular cAMP levels in NHBE cells.

MeSH terms

  • Bronchi / cytology
  • Bronchi / metabolism*
  • Cells, Cultured
  • Chlorides / metabolism*
  • Cyclic AMP / metabolism
  • Dose-Response Relationship, Drug
  • Epithelial Cells / metabolism
  • Humans
  • Vasoactive Intestinal Peptide / metabolism*

Substances

  • Chlorides
  • Vasoactive Intestinal Peptide
  • Cyclic AMP