The frequency of phospholipase A2 binding of basophilic granulocytes does not decrease during bee-venom-specific immunotherapy

Allergy. 1999 Jul;54(7):742-7. doi: 10.1034/j.1398-9995.1999.00952.x.

Abstract

Background: The major allergenic component of bee venom is phospholipase A2 (PLA2).

Methods: In this study, PLA2 was used to analyze and enrich PLA2-binding cells from peripheral blood by high gradient magnetic cell sorting.

Results: In normal donors, the frequency of allergen (PLA2)-binding cells among peripheral blood mononuclear cells (PBMC) as determined by flow cytometry is below 0.1%, whereas in bee-venom-allergic patients, PLA2-binding cells are readily detectable at frequencies of up to 2.3%. In severely bee-venom-allergic patients, many basophilic granulocytes are present, as defined by anti-CD9, CD25, and CD38 mAb, comprising up to 95% of the PLA2-binding cells. From blood of allergic and normal donors, about equal absolute numbers of allergen-binding CD19/21-positive B cells can be enriched. Severe anaphylactic reactions (Mueller grade IV) and failure of or adverse reactions during immunotherapy are associated with high numbers of circulating allergen-binding basophils. Interestingly, in the patients studied, the number of PLA2-binding basophilic granulocytes did not markedly change during rush immunotherapy and up to 6 months of maintenance immunotherapy.

Conclusions: The specific and reproducible enrichment of PLA2-binding cells provides a new tool for the analysis and monitoring of effector cells in bee-venom-allergic patients with immediate-type hypersensitivity.

MeSH terms

  • Adolescent
  • Adult
  • Basophils / cytology*
  • Basophils / metabolism*
  • Bee Venoms / administration & dosage
  • Bee Venoms / immunology*
  • Desensitization, Immunologic*
  • Flow Cytometry / methods
  • Humans
  • Hypersensitivity, Immediate / therapy*
  • Immunomagnetic Separation
  • Leukocyte Count
  • Phospholipases A / metabolism*
  • Phospholipases A2
  • Skin Tests

Substances

  • Bee Venoms
  • Phospholipases A
  • Phospholipases A2