Disappearance of gastric antral vascular ectasia after percutaneous transhepatic obliteration

Hepatogastroenterology. 1999 Jan-Feb;46(25):185-8.

Abstract

We report on a patient with severe iron deficiency anemia due to gastric antral vascular ectasia (GAVE), complicating hepatocellular carcinoma (HCC) and esophageal varices. Esophago-gastro-duodenoscopy (EGDS) 6 months after transarterial embolization (TAE) for the HCC and percutaneous transhepatic obliteration (PTO) for esophageal varices, showed the absence of GAVE. As GAVE did not recur in spite of the recurrence of the tumor thrombus later, lowered antral congestion by PTO might be the main cause of disappearance of GAVE. This case suggests that PTO may be an effective treatment against GAVE with portal hypertension with uncontrollable bleeding.

Publication types

  • Case Reports

MeSH terms

  • Aged
  • Carcinoma, Hepatocellular / complications
  • Carcinoma, Hepatocellular / therapy
  • Embolization, Therapeutic
  • Esophageal and Gastric Varices / complications
  • Gastric Antral Vascular Ectasia / complications
  • Gastric Antral Vascular Ectasia / therapy*
  • Humans
  • Hypertension, Portal / complications
  • Liver Neoplasms / complications
  • Liver Neoplasms / therapy
  • Male
  • Neoplasm Recurrence, Local
  • Stents